Traumatic optic neuropathy: Difference between revisions

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==Clinical Features==
==Clinical Features==
===History===
===History===
*Acute unilateral vision loss, decreased color sensation, and/or visual field deficit after blunt or penetrating trauma
*Acute unilateral [[vision loss]], decreased color sensation, and/or [[visual field defects|visual field deficit]] after blunt or penetrating trauma
*Complaints may be delayed due to the impact of and treatment for other concomitant head injuries  
*Complaints may be delayed due to the impact of and treatment for other concomitant head injuries  


===Physical Exam===
===Physical Exam===
*External eye exam may show signs of orbital trauma or fracture (soft tissue edema, hematoma, step-off on palpation of orbital rim)
*External eye exam may show signs of [[orbital trauma]] or [[orbital fracture|fracture]] (soft tissue edema, hematoma, step-off on palpation of orbital rim)
*Decreased visual acuity and an afferent pupillary defect (in unilateral cases)
*[[vision loss|Decreased visual acuity]] and an afferent pupillary defect (in unilateral cases)
*On funduscopy, the initial optic nerve head assessment will be normal. Optic atrophy may be seen 3-6 weeks after trauma.
*On [[fundoscopy]], the initial optic nerve head assessment will be normal; optic atrophy may be seen 3-6 weeks after trauma


==Differential Diagnosis==
==Differential Diagnosis==
Posterior ischemic optic neuropathy<br>Optic neuritis<br>Optic nerve avulsion<br>Non-organic vision loss<br>Pre-/intra-/subretinal hemorrhage<br>Choroidal Rupture <br>Commotio retinae<br>
*Posterior ischemic optic neuropathy
*[[Optic neuritis]]
*Optic nerve avulsion
*Non-organic vision loss
*Pre-/intra-/sub[[retinal hemorrhage]]
*Choroidal rupture
*Commotio retinae
*[[Traumatic iritis]]
 
{{Acute vision loss noninflamed DDX}}
 
{{Eye trauma}}


==Evaluation==
==Evaluation==
===Workup===
===Workup===
*Non-contrast head and face (including orbits) CT
*Non-contrast CT of head and face (including orbits)


===Diagnosis===
===Diagnosis===
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==External Links==
==External Links==
*[[http://eyewiki.aao.org/Traumatic_Optic_Neuropathy]]
*http://eyewiki.aao.org/Traumatic_Optic_Neuropathy


==References==
==References==
<references/>
<references/>
[[Category:Ophthalmology]]
[[Category:Trauma]]

Latest revision as of 21:49, 6 October 2019

Background

  • Acute injury to the optic nerve from direct or indirect trauma resulting in vision loss
  • Caused by indirect injury to the optic nerve (intracanalicular portion) from transmitted shock from an orbital impact
  • Can also be caused by directly by penetrating injury or from bony fragments in the optic canal or orbit, or orbital hemorrhage and optic nerve sheath hematoma

Pathophysiology

  • Indirect: Hypothesized to result from shearing injury to the intracanalicular portion of optic nerve, which can cause axonal injury or disturb the blood supply of the optic nerve
  • Direct: Presumed to be the result of tissue disruption secondary to foreign body or bony fragments impacting the optic nerve

Clinical Features

History

  • Acute unilateral vision loss, decreased color sensation, and/or visual field deficit after blunt or penetrating trauma
  • Complaints may be delayed due to the impact of and treatment for other concomitant head injuries

Physical Exam

  • External eye exam may show signs of orbital trauma or fracture (soft tissue edema, hematoma, step-off on palpation of orbital rim)
  • Decreased visual acuity and an afferent pupillary defect (in unilateral cases)
  • On fundoscopy, the initial optic nerve head assessment will be normal; optic atrophy may be seen 3-6 weeks after trauma

Differential Diagnosis

Acute Vision Loss (Noninflamed)

Emergent Diagnosis

Orbital trauma

Acute

Subacute/Delayed

Evaluation

Workup

  • Non-contrast CT of head and face (including orbits)

Diagnosis

  • Primarily a clinical diagnosis

Management

  • Ophthalmology consult
    • Generally, no acute interventions are known to change prognosis

Contraindicated

  • Corticosteroids not helpful[1]
  • Surgical intervention has not shown to be beneficial, although some support the use of surgery in certain scenarios (e.g. bony fragment abutting optic nerve or optic nerve sheath hematoma)

Disposition

  • Outpatient ophthalmology follow up

Prognosis

  • Visual improvement in only about 50% of patients

See Also

External Links

References

  1. Levin, L.A., et al., The treatment of traumatic optic neuropathy: the International Optic Nerve Trauma Study. Ophthalmology, 1999. 106(7): p. 1268-77.