Undifferentiated shock: Difference between revisions
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== Undifferentiated Hypotension Algorithm == | == Undifferentiated Hypotension Algorithm == | ||
*Which of the following is the main cause? | |||
*Which of the following is contributing? | |||
Check the following in order: | |||
#HR (assess based on pt's age) | |||
##Too slow or too fast? (to the point where CO is affected) | |||
###If so HR is likely primary etiology of hypotension | |||
###Pace or cardiovert | |||
#Volume Status | |||
##What is the LV end-diastolic volume? | |||
###Approximated by the CVP, IVC diameter | |||
##If low must rule-out occult blood loss: | |||
###FAST for intra-abdominal source | |||
###US to rule-out AAA | |||
###Guaiac to rule-out GI bleed | |||
###CXR to rule-out hemothorax | |||
#Contractility | #Contractility | ||
## | ##Is the myocardium severely decreased in its contractile function? | ||
## | ###Poor contractility on ultrasound, bounding/thready pulse, hyperdynamic precordium | ||
##Is forward flow occurring? | |||
### | ###Assess for valvular dysfunction (MR, AR) | ||
###Assess for obstruction (PE, HOCM) | |||
### | #Systemic Vascular Resistance | ||
##Pathologic vasodilation (decreased SVR) suggested by: | |||
###Warm extremities | |||
###Bounding pulse | |||
# | ###The other three components are normal (HR, volume status, contractility) | ||
## | |||
## | |||
# | |||
# | |||
## | |||
### | |||
== Lack of Response to Normal Tx (DDX) == | == Lack of Response to Normal Tx (DDX) == | ||
Revision as of 02:53, 7 May 2012
Definition
- SBP <90 in normal pt
- SBP <100 with h/o HTN or age >60
- Lactate > 4 or base def < -4
Types
| Type | Skin | HR | Oth |
| Hypovolemic | cold | inc | |
| Obstructive | cold | inc | |
| Cardiogenic | cold | inc/dec | ?dysth |
| Distributive | warm | inc | |
| Neurogenic | warm | dec |
Undifferentiated Hypotension Algorithm
- Which of the following is the main cause?
- Which of the following is contributing?
Check the following in order:
- HR (assess based on pt's age)
- Too slow or too fast? (to the point where CO is affected)
- If so HR is likely primary etiology of hypotension
- Pace or cardiovert
- Too slow or too fast? (to the point where CO is affected)
- Volume Status
- What is the LV end-diastolic volume?
- Approximated by the CVP, IVC diameter
- If low must rule-out occult blood loss:
- FAST for intra-abdominal source
- US to rule-out AAA
- Guaiac to rule-out GI bleed
- CXR to rule-out hemothorax
- What is the LV end-diastolic volume?
- Contractility
- Is the myocardium severely decreased in its contractile function?
- Poor contractility on ultrasound, bounding/thready pulse, hyperdynamic precordium
- Is forward flow occurring?
- Assess for valvular dysfunction (MR, AR)
- Assess for obstruction (PE, HOCM)
- Is the myocardium severely decreased in its contractile function?
- Systemic Vascular Resistance
- Pathologic vasodilation (decreased SVR) suggested by:
- Warm extremities
- Bounding pulse
- The other three components are normal (HR, volume status, contractility)
- Pathologic vasodilation (decreased SVR) suggested by:
Lack of Response to Normal Tx (DDX)
- Cardiogenic
- Acute Valvular Regurg/VSD
- CHF
- Dysrhythmia
- Ischemia/Infarction
- Myocardial Contusion/Myocarditis
- Obstructive
- Air embolism
- Aortic Stenosis
- Cardiac Tamponade
- Massive PE
- Tension Pneumo
- Distributive
- Adrenal Crisis
- Anaphylaxis
- Neurogenic
- Sepsis
- Toxicologic
- Hypovolemic
- Hemorrhage Traumatic and Non-traumatic
- Severe Dehydration
See Also
Source
2/06 DONALDSON (Adapted from Tintinalli)
Morchi 2010
