Hyperaldosteronism: Difference between revisions
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==Background== | ==Background== | ||
Hyperaldosteronism refers to a condition of excess aldosterone secretion, typically leading to sodium retention, potassium excretion, and metabolic alkalosis. It is categorized into: | |||
* Primary hyperaldosteronism (Conn syndrome) – autonomous overproduction of aldosterone, most commonly from an adrenal adenoma or bilateral adrenal hyperplasia. | |||
* | |||
* Secondary hyperaldosteronism – due to increased renin from conditions like renal artery stenosis, heart failure, or cirrhosis. | |||
Primary hyperaldosteronism is an important and potentially reversible cause of secondary hypertension, accounting for 5–10% of hypertensive cases and up to 20% of treatment-resistant hypertension. It is often underdiagnosed in emergency settings. | |||
==Clinical Features== | ==Clinical Features== | ||
Revision as of 23:41, 5 May 2025
Background
Hyperaldosteronism refers to a condition of excess aldosterone secretion, typically leading to sodium retention, potassium excretion, and metabolic alkalosis. It is categorized into:
- Primary hyperaldosteronism (Conn syndrome) – autonomous overproduction of aldosterone, most commonly from an adrenal adenoma or bilateral adrenal hyperplasia.
- Secondary hyperaldosteronism – due to increased renin from conditions like renal artery stenosis, heart failure, or cirrhosis.
Primary hyperaldosteronism is an important and potentially reversible cause of secondary hypertension, accounting for 5–10% of hypertensive cases and up to 20% of treatment-resistant hypertension. It is often underdiagnosed in emergency settings.